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추천 연관자료
- 목차
-
1. Akinetopsia
-Definition
-Cause
-Case study(L.M) (1. 1983 / 2. 1995 )
-Other case
2. Blindsight
-Definition & Cause
-Case study – (Case 1. Helen / B. Case 2. Patient D.B)
-Two visual systems
-Affective blindsight
-Explaining blindsight
3. Visual Agnosia
-Definition & Cause / Two types of Visual Agnosia
-Case study (Patient D.F)
- 본문내용
-
Case Study - L.M. Ⅰ
SELECTIVE DISTURBANCE OF MOVEMENT VISION AFTER BILATERAL BRAIN DAMAGE
(S. Zihl et al, 1983)
Movement Vision
Horizontal and vertical motion
Question
- Clear Motion
- Unclear Motion
- No Motion
(중략)
Case II. Patient D.B.(Weiskrantz, 1974)
Patient D.B. : 34 years old , male
오른쪽 primary visual cortex (V1). 정맥성 종양
“오른쪽 1차 시각피질” 절제
(26 years old)
왼쪽 대측 시야 손상.
- 참고문헌
-
A. Charnallet et al.(2008). Associative visual agnosia: A case study. Behavioural Neurology 19, 41-44
A.D.Milner et al.(1991). Perception and Action in ‘Visual Form Agnosia’. Brain, 114, 405-428
Beatrice de Gelder and Marco Tamietto (2007). Scholarpedia, 2(10): 3555.
Beckers G. and S. Zeki (1995) The consequences of inactivating areas V1 and V5 on visual motion perception. Brain 118, 49-60.
B Kolb & I.Q. Whishow (1985). Fundamentals of Human Neuropsychology (2.edition).WH Freeman, NY.
C. Temple (1997). Developmental Cognitive Neuropsychology. Psychology Press, Ltd.
Cowey, A., & Stoerig, P. (1989). Projection patterns of surviving neurons in the dorsal lateral geniculate nucleus following discrete lesions of striate cortex: implications for residual vision. Experimental Brain Research, 75(3), 631-638.
de Gelder, B., Morris, J. S., & Dolan, R. J. (2005). Unconscious fear influences emotional awareness of faces and voices. Proceedings of the National Academy of Sciences, U.S.A., 102, 18682-18687.
de Gelder, B., Vroomen, J., Pourtois, G., & Weiskrantz, L. (1999). Non-conscious recognition of affect in the absence of striate cortex. Neuroreport, 10, 3759-3763.
Fendrich, R., Wessinger, C. M., & Gazzaniga, M. S. (1992). Residual vision in a scotoma: implications for blindsight. Science, 258(5087), 1489-1491.
Gazzaniga, M. S., Fendrich, R., & Wessinger, C. M. (1994). Blindsight reconsidered. Current Directions in Psychological Science, 3(3), 93-96.
Gazzaniga, M. S., Eliassen, J. C., Nisenson, L., Wessinger, C. M., Fendrich, R., & Baynes, K. (1996). Collaboration between the hemispheres of a callosotomy patient Emerging right hemisphere speech and the left hemisphere interpreter. Brain, 119(4), 1255-1262.
Hendry, S. H., & Reid, R. C. (2000). The koniocellular pathway in primate vision. Annual review of neuroscience, 23(1), 127-153.
Humprey. N. K., Weiskrantz. L. (1967). Vision in Monkeys after Removal of the Striate Cortex. Nature, vol.215, 595-597
K. Heilman & E. Valenstein (Eds) 2003. Clinical Neuropsychology (4.edition). Oxford Univ.press.
Matthew Rizzo, Mark Nawrot, Josef Zihl (1995). Motion and shape perception in cerebral akinetopsia. Brain 118, 1105-1127.
M.A. Goodale & A. D. Milner (2004). Sight Unseen: An Exploration of Conscious and Unconscious Vision. 2004, Oxfor Univ. Press, NY
Mc Carthy & Warrington(1986). Visual associative agnosia: a clinico-anatomical study of a single case. Journal of Neurology, Neurosurgery, and Psychiatry; 49:1233-1240
Morland, A. B., Lê, S., Carroll, E., Hoffmann, M. B., & Pambakian, A. (2004). The role of spared calcarine cortex and lateral occipital cortex in the responses of human hemianopes to visual motion. Journal of cognitive neuroscience,16(2), 204-218.
Paul A. Shelton et al.(1994). Apperceptive Visual Agnosia: A Case Study. Brain and Cognition 25, 1-23
Pelak, Victoria S., William F. Hoyt (2005). Symptoms of akinetopsia associated with traumatic brain injury and Alzheimer's Disease. Neuro-Ophthalmology 29, 137-142.
Pierre Jacob(2005). Grasping and Perceiving Objects. Cognition and the Brain, 241-283
Murray Grossman&Steven Galetta&Mark D’esposito(1997), Object Recognition Difficulty in Visual Apperceptive Agnosia, Brain and Cognition 33, 306-342
Schmid, M. C., Mrowka, S. W., Turchi, J., Saunders, R. C., Wilke, M., Peters, A. J., & Leopold, D. A. (2010). Blindsight depends on the lateral geniculate nucleus. Nature, 466(7304), 373-377.
Shaun P. Vecera&Kendra S. Gilds(1998). What Processing Is Impaired in Apperceptive Agnosia?: Evidence from Normal Subjects. Journal of Cognitive Neuroscience 10:5, pp.568-580
Shipp, S., B.M. de Jong, J. Zihl, R.S.J. Frackowiak, and S. Zeki (1994). The brain activity related to residual motion vision in a patient with bilateral lesions of V5. Brain 117, 1023-1038.
Sincich, L. C., Park, K. F., Wohlgemuth, M. J., & Horton, J. C. (2004). Bypassing V1: a direct geniculate input to area MT. Nature neuroscience,7(10), 1123-1128.
Weiskrantz. L., Warrington E. K., Sanders M.D., Marshall. J. (1974). Visual capacity in the hemianopic field following a restricted occipital ablation, Brain, 97, 709-728
Weiskrantz. L., Cowey. A., Mare. L. M. (1998). Learning from the pupil: a spatial visual channel in the absence of V1 in monkey and human, Brain, 121, 1065-1072
Zeki, Semir (1991). Cerebral akinetopsia (visual motion blindness): A review. Brain 114, 811-824.
Zihl, J, D von Cramon, N Mai (1983). Selective disturbance of movement vision after bilateral brain damage. Brain 106, 313-340.
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