[한의학][논문요약] Smoking is negatively associated with the presence of thyroglobulin autoantibody and to a lesser deg

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The expression and regulation of VEGF on nicotine and lipopolysaccharide-stimulated PDL cellIntroduction1. Effects of nicotine and lipopolysaccharide on periodontal ligament2. VEGF1) VEGF2) VEGF up-regulation & down-regulation3. SIRT4. MMP series5. VEGF signaling PathwayEffects of smoking on periodontal diseaseCurrent and long-term smoking in young adults is detrimental to periodontal health, but smoking cessation may be associated with a relatively rapid improvement in the periodontium.Thomson WM, Broadbent JM, Welch D, Beck JD, Poulton R Cigarette smoking and periodontal disease

The objective of this current research was to consolidate these factors and determine their level of influence, either positively or negatively from the adopter’s perspective, on the adoption decision. 이연구의 목적은 이러한 요인(factor)들을 통합하고 각각의 영향의 정도(level)를 도입자의 관점에서 장단점을 규정하는데 있다. A set of 26 factors, used as variables in various adoption models from the literature, were presented in a neutral manner, without pre-classifying them as barriers or incentives, through a survey sent to SMEs. 논문의 다양

of vasopressin (antidiuretic hormone)④ suppressing atrial natriuretic peptide release.The primary renal salt and water retention may also contribute to edema formation in some cases.② Hyperlipidemiais believed to be a consequence of increased hepatic lipoprotein synthesis that is triggered by reduced oncotic pressure and may be compounded by increased urine loss of proteins that regulate lipid homeostasis. Low-density lipoproteins and cholesterol are increased in the majority of patients, whereas very low density lipoproteins and triglycerides tend to rise in patients with severe dis

of vasopressin (antidiuretic hormone)④ suppressing atrial natriuretic peptide release.The primary renal salt and water retention may also contribute to edema formation in some cases.② Hyperlipidemiais believed to be a consequence of increased hepatic lipoprotein synthesis that is triggered by reduced oncotic pressure and may be compounded by increased urine loss of proteins that regulate lipid homeostasis. Low-density lipoproteins and cholesterol are increased in the majority of patients, whereas very low density lipoproteins and triglycerides tend to rise in patients with severe dis